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Brain circuits may explain why opposite obesity drugs both work
Key Takeaways
- Cambridge researchers used mice to explain why opposite GIPR drug strategies can both promote weight loss.
- The study found that GIPR stimulation in the brainstem and GIPR blockade in the hypothalamus can produce similar outcomes.
- The results may help researchers design more effective obesity drug combinations with GLP-1-based treatments.
- The findings clarify mechanism, but the evidence described here is still preclinical rather than human clinical proof.
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DT Editorial Team··via medicalxpress.com
