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Researchers found a new way to strip drug-resistant tumors of a key DNA repair advantage
Key Takeaways
- Researchers identified UNI418 as a molecule that reduces key DNA repair proteins in cancer cells.
- The compound appears to work by activating the Cul4A ubiquitin ligase degradation pathway.
- The finding could point to a new strategy for overcoming resistance to DNA-damage-based cancer therapies.
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DT Editorial Team··via medicalxpress.com